Ofloxacin is a synthetic, broad spectrum first generation bactericidal fluoroquinolone.
Its empirical formula is C18H20FN3O4, and its molecular weight is 361.4.The chemical structure is:
Ofloxacin inhibits the enzyme bacterial DNA gyrase and prevents replication of bacterial DNA during bacterial growth and reproduction.
Nearly 100% of the orally administered dose is absorbed. Food delays rate of absorption of ofloxacin but do not significantly affect the extent of absorption.
Ofloxacin is active against many gram-positive bacteria and gram-negative bacteria. Relatively long post-antibiotic effect. Ofloxacin has intermediate activity between norfloxacin and ciprofloxacin for gram-negative bacteria. It has better activity against gram-positive organism and further better against chlamydia and mycoplasma compared to ciprofloxacin.
Condition | Dosage (adults) |
Acute Bacterial Exacerbation of Chronic Bronchitis | 400 mg 12 hourly for 10 days |
Comm. Acquired Pneumonia | 400 mg 12 hourly for 10 days |
Uncomplicated Skin and Skin Structure Infections | 400 mg 12 hourly for 10 days |
Acute, Uncomplicated Urethral and Cervical Gonorrhoea | 400 mg single dose |
Non-gonococcal Cervicitis/Urethritis due to C. trachomatis | 300 mg 12 hourly for 7 days |
Mixed Infection of the urethra and cervix due to C. trachomatis and N. gonorrhoeae | 300 mg 12 hourly for 7 days |
Acute Pelvic Inflammatory Disease | 400 mg 12 hourly for 10-14 days |
Uncomplicated Cystitis due to E. coli or K. pneumonia | 200 mg 12 hourly for 3 days |
Uncomplicated Cystitis due to other approved pathogens | 200 mg 12 hourly for 7 days |
Complicated UTI's | 200 mg 12 hourly for 10 days |
Prostatitis due to E.Coli | 300 mg 12 hourly for 6 weeks |
Patients receiving ofloxacin should be well hydrated to prevent the formation of highly concentrated urine so that crystalluria does not occur. Ofloxacin is contraindicated in patients with known hypersensitivity to ofloxacin or any other fluoroquinolone and in patients with QTc prolongation.
Side effects of norfloxacin are dizziness, nausea, headache, abdominal cramps, anorexia, diarrhea, constipation, dyspepsia, flatulence, tingling of the fingers, vomiting, tendonitis, tendon repture, liver toxicity and kidney toxicity.
Ofloxacin is a synthetic, broad spectrum first generation fluoroquinolone.
Its empirical formula is C18H20FN3O4, and its molecular weight is 361.4.The chemical structure is:
Oral: 200, 300, 400 mg tablets
Ophthalmic : 3 mg/mL solution
Otic : 0.3% solution
Ofloxacin like other fluoroquinolones (FQs) inhibits the enzyme bacterial DNA gyrase that nicks double-stranded DNA, introduces negative supercoils and then reseals the nicked ends. This is essential to avert excessive positive supercoiling of the strands when they separate to permit replication or transcription. The DNA gyrase consists of two A and two B subunits: The A subunit brings about nicking of DNA, B subunit introduces negative supercoils and then A subunit reseals the strands. Ofloxacin binds to A subunit with high affinity and restricts its strand cutting and resealing function. In gram-positive bacteria the major target of action is an analogous enzyme topoisomerase IV which nicks and separates daughter DNA strands once the DNA replication is complete. The damaged DNA leads to formation of exonucleases resulting in digestion of the DNA and this possibly contributes to the bactericidal action of ofloxacin.
The mammalian cells possess an enzyme topoisomerase II instead of DNA gyrase or topoisomerase IV that has very low affinity for ofloxacin - thus the low toxicity to host cells.
Aerobic gram-positive microorganisms
Aerobic gram-negative microorganisms
Like other fluoroquinolones, some strains of Pseudomonas aeruginosa may develop resistance fairly rapidly during treatment with ofloxacin.
Other microorganisms
Ofloxacin is not active against Treponema pallidum.
Various strains of other streptococcal species, Enterococcus species, and anaerobes are resistant to ofloxacin.
Like other fluoroquinolones, resistance is mainly because of chromosomal mutation (Quinolone-Resistance Determining Regions {QRDRs}) forming a DNA gyrase or topoisomerase IV with reduced affinity for ofloxacin. Another common mechanism is reduced permeability/increased efflux of ofloxacin across bacterial membranes. Like other FQs ofloxacin, FQ-resistant mutants are not easily selected hence resistance develops slowly to FQs.
Due to the unique mechanism of action of fluoroquinolones plasmid mediated transferable resistance perhaps does not occur.
Recently two types of plasmid-mediated resistance have been reported.
Both mechanisms confer low-level resistance which may facilitate the point mutations that confer high-level resistance. Resistance to one fluoroquinolone, particularly if it is of high level, usually confers cross-resistance to all other members of the class.
Resistance to ofloxacin due to spontaneous mutation in vitro is rare.
Ofloxacin is rapidly and well absorbed orally and bioavailability is almost 100%.The peak plasma concentration is attained 1 to 2 hours after an oral doseof 400 mg. Absorption may be delayed by the presenceof food, but the extent of absorption is not substantiallyaffected. Plasma protein binding is approximately 25%. Ofloxacin isextensively distributed in body fluids, including the CSF,and tissue penetration is good. It crosses the placentaand is distributed into breast milk. It also appears in thebile. The elimination of ofloxacin is biphasic; half-lives ofabout 4 to 5 and 20 to 25 hours have been reported forthe 2 phases, respectively. In renal impairment, the t1/2 increases to about15 to 60 hours. There is limitedmetabolism to desmethyl and N-oxide metabolites and desmethylofloxacin has moderate antibacterial activity. Ofloxacin is excreted primarily by the kidneys by tubular secretion and glomerular filtration. About 65 to 80% of a dose is eliminated unchanged in theurine over 24 to 48 hours, resulting in high urinary concentrations. Less than 5% is excreted in the urine asmetabolites. About 4 to 8% of a dose may be eliminatedin the faeces.
Condition | Dosage (adults) |
Acute Bacterial Exacerbation of Chronic Bronchitis | 400 mg 12 hourly for 10 days |
Comm. Acquired Pneumonia | 400 mg 12 hourly for 10 days |
Uncomplicated Skin and Skin Structure Infections | 400 mg 12 hourly for 10 days |
Acute, Uncomplicated Urethral and Cervical Gonorrhoea | 400 mg single dose |
Non-gonococcal Cervicitis/Urethritis due to C. trachomatis | 300 mg 12 hourly for 7 days |
Mixed Infection of the urethra and cervix due to C. trachomatis and N. gonorrhoeae | 300 mg 12 hourly for 7 days |
Acute Pelvic Inflammatory Disease | 400 mg 12 hourly for 10-14 days |
Uncomplicated Cystitis due to E. coli or K. pneumoniae | 200 mg 12 hourly for 3 days |
Uncomplicated Cystitis due to other approved pathogens | 200 mg 12 hourly for 7 days |
Complicated UTI's | 200 mg 12 hourly for 10 days |
Prostatitis due to E.Coli | 300 mg 12 hourly for 6 weeks |
All the conditions mentioned in the above table are due to susceptible organisms stated in the microbiology section
ofloxacin and cyclosporine has not been studied.
The frequency or severity of adverse reactions was not different in elderly adults compared with younger adults treated with ofloxacin. Also the pharmacokinetic properties were similar. Dosage adjustment is necessary for elderly patients with impaired renal function
It is a pregnancy Category C drug. Ofloxacin has not shown any teratogenic effect in most of the animal species tested. However well-controlled studies in pregnant women have not been conducted. Ofloxacin should be used during pregnancy only if the potential benefit justifies the potential risk to the foetus.
Ofloxacin is excreted in human milk with concentration same as in plasma.Hence they should be avoided during lactation.
The safety and effectiveness of oral ofloxacin in paediatric patients and adolescents below the age of 18 years have not been established. Ofloxacin causes arthropathy in juvenile animals of several animal species.
In patients with impaired renal function (creatinine clearance <50 mg/mL), modification of the dosage regimen is mandatory.
In patients with known or suspected hepatic insufficiency/impairment, careful clinical observation and appropriate laboratory studies should be performed before and during therapy since excretion of ofloxacin may be reduced.
Long-term carcinogenicity studies in rats and mice resulted in no carcinogenic or tumorigenic effects due to ofloxacin. Also no adverse effects were observed in reproduction studies.
Ofloxacin is contraindicated in persons with a history of hypersensitivity associated with the use of ofloxacin or any member of the quinolone group of antimicrobial agents.
The commonly seen adverse effects are:
Less commonly seen adverse effects were: